BPA Worse After Metabolism

Written by Dr. Andrea Maxim, Naturopathic Doctor

Measured through urine, it has been shown that over 82% of American’s are already exposed to toxic levels of bisphenol A (BPA), and why not? We are chronically exposed to BPA as it is found in any malleable plastic such as water bottles, Saran wrap, food and beverage can linings, toilet paper, dental amalgams and even thermal sales receipts.

When metabolized by the body, BPA has a similar structure to estrogen thus allowing it to bind to the estrogen receptors causing many adverse health effects: estrogen-related cancers, neurological disorders, obesity and hormone disruption. However, research has found that the binding of BPA is weak but the metabolite (breakdown product) of BPA called MBP may bind to the estrogen receptors anywhere from 100-fold to 1000-fold stronger than BPA itself. The reasoning for this has not yet been determined but researches suspect that it could be from the greater length of the MBP molecule allowing both ends to interact with the receptor, much like biological estrogen.

12 SIMPLE SNACKS RECIPES BELOW

I try to educate all my patients on a condition called estrogen dominance. Every man, woman, child and animal are toxic in environmental estrogens which I believe is causing early development in females, hair-thinning in young males and many of the infertility and hormone-related conditions being suffered by millions.

If you are interested in discussing how to protect yourself from these harmful estrogens and perhaps starting a detox program to rid the body of toxins already exposed, please do not hesitate to contact Dr. Andrea Maxim, Naturopathic Doctor Now!!

Andrea Maxim, ND – Creator of the MAXIM MOVEMENT

www.facebook.com/andrea.maximnd
www.healingjourneynaturopathic.com (Caledonia Clinic)
Twitter: @AndreaMaximND
YouTube: NaturopathicHealing
P: (888) 375 3111

References:

Michael E. Baker, Charlie Chandsawangbhuwana. 3D Models of MBP, a Biologically Active Metabolite of Bisphenol A, in Human Estrogen Receptor α and Estrogen Receptor β. PLoS ONE, 2012; 7 (10): e46078 DOI: 10.1371/journal.pone.0046078

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